Tranexamic Acid Txhaj

Nthuav Qhia:

2ml: 0.2g

10amps / thawv

Txhawb OEM / ODM:Yog

MOQ:500,000 Amps

Tus me nyuam lub sij hawm:30 ~ 55 Hnub

Kev Luam Tshaj:FOB, CIF

Payment Term:T / T, L / C

Standard Muaj:CP, PEB

Cov ntaub ntawv:GMP, CPP, FSC, CTD

Kev tshwm sim

1. Cov khoom no feem ntau yog siv rau txhua hom los ntshav vim mob hnyav lossis mob ntev, txwv lossis kab mob thawj fibrinolysis. Nws feem ntau tsis siv nyob rau hauv theem nrab hyperfibrinolytic xeev tshwm sim los ntawm kev nthuav tawm intravascular coagulation ua ntej heparinization.

2. Rau kev raug mob los yog kev phais los ntshav nyob rau hauv cov kab mob fibrinogen-nplua nuj xws li prostate, urethra, ntsws, hlwb, uterus, qog adrenal thiab thyroid.

3. Siv los ua tus antagonist ntawm cov nqaij mos-type fibrinogen activator (t-PA), streptokinase thiab urokinase.

4. Rau fibrinolytic los ntshav vim nchuav menyuam, thaum ntxov placental abruption, stillbirth thiab amniotic kua embolism, thiab muaj zog menorrhagia vim localized fibrinolytic lesions nyob rau hauv lub uterine kab noj hniav.

Cov kev phiv tshuaj

1. Qee zaum, muaj intracranial thrombosis thiab los ntshav vim yog siv tshuaj ntau dhau.

2.Tej zaum yuav muaj raws plab, xeev siab, thiab ntuav.

3.Tsis tshua muaj, muaj kev coj khaub ncaws tsis zoo (vim cov ntshav txhaws thaum cev xeeb tub).4 Vim tias cov khoom tuaj yeem nkag mus rau hauv cov kua cerebrospinal, cov tsos mob hauv nruab nrab ntawm lub paj hlwb xws li qhov muag tsis pom kev, mob taub hau, kiv taub hau, thiab qaug zog tuaj yeem tshwm sim tom qab txhaj tshuaj, tshwj xeeb tshaj yog nyob rau hauv kev cuam tshuam. tus nqi txhaj tshuaj, tab sis tsis tshua muaj.

CIA

Khaws rau hauv qhov chaw txias, qhuav thiab tsaus.

Khaws txhua yam tshuaj tawm ntawm menyuam yaus.